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doi: 10.1172/JCI169922 71 KirklandJTchkoniaT
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It inhibited NO, PGE-2 and proinflammatory cytokines (TNF-, IL-1 and IL-6) while reducing ROS production and NADPH oxidase activation
This increased transmission of pulsatile and high-pressure blood flow into the cerebral circulation is associated with cerebral microvascular damage, which contributes to chronic reductions in CBF seen with aging and disease (Suri et al., 2020)
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